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Nicotinamide enhances repair of arsenic and ultraviolet radiation-induced DNA damage in HaCaT keratinocytes and ex vivo human skin.

机译：烟酰胺增强了HaCaT角质形成细胞和离体人体皮肤中砷和紫外线辐射诱导的DNa损伤的修复。

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Benjamin C Thompson; Gary M Halliday; Diona L Damian;
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1. Nicotinamide enhances repair of ultraviolet radiation-induced DNA damage in human keratinocytes and ex vivo skin [J] . SurjanaD., HallidayG.M., DamianD.L. Carcinogenesis . 2013,第5期

机译：烟酰胺可增强紫外线对人角质形成细胞和离体皮肤的DNA损伤的修复作用
2. Protective Effect of Diphlorethohydroxycarmalol against Ultraviolet B Radiation-Induced DNA Damage by Inducing the Nucleotide Excision Repair System in HaCaT Human Keratinocytes [J] . Hee Kyoung Kang, Jin Won Hyun, Kyoung Ah Kang, Marine Drugs . 2015,第9期

机译：邻苯三酚对人角质形成细胞中核苷酸切除修复系统的紫外线B辐射诱导的DNA损伤的保护作用。
3. Nicotinamide enhances repair of ultraviolet radiation-induced DNA damage in primary melanocytes [J] . ThompsonB.C., SurjanaD., HallidayG.M., Experimental dermatology . 2014,第7期

机译：烟酰胺可增强紫外线对原代黑素细胞DNA损伤的修复作用
4. Influence of non-thermal atmospheric pressure plasma on DNA of human keratinocytes (HaCaT): Role of reactive oxygen species [C] . Blackert S., Haertel B., Lindequist U., 2011 Abstracts IEEE International Conference on Plasma Science . 2011

机译：非热大气压血浆对人角质形成细胞（HaCaT）DNA的影响：活性氧的作用
5. Cigarette smoke, DNA damage and repair in human cervical and vaginal cells in vitro and ex vivo [D] . Moktar, Afsoon 2010

机译：体外和离体的香烟烟雾，DNA损伤和人类宫颈和阴道细胞的修复
6. Nicotinamide Enhances Repair of Arsenic and Ultraviolet Radiation-Induced DNA Damage in HaCaT Keratinocytes and Ex Vivo Human Skin [O] . Benjamin C. Thompson, Gary M. Halliday, Diona L. Damian -1

机译：烟酰胺增强HaCaT角质形成细胞和离体人体皮肤中砷和紫外线辐射诱导的DNA损伤的修复。
7. Protective Effect of Diphlorethohydroxycarmalol against Ultraviolet B Radiation-Induced DNA Damage by Inducing the Nucleotide Excision Repair System in HaCaT Human Keratinocytes [O] . Mei Jing Piao, Susara Ruwan Kumara Madduma Hewage, Xia Han, 2015

机译：二氯乙氧基卡波林诱导HaCaT人角质形成细胞核苷酸切除修复系统对紫外线B辐射诱导的DNa损伤的保护作用

Nicotinamide enhances repair of arsenic and ultraviolet radiation-induced DNA damage in HaCaT keratinocytes and ex vivo human skin.

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